Case study · 6 questions
Heart Failure NCLEX Case Study: 6 Next Gen Questions
An NCLEX heart failure (CHF) case study set on a medical-surgical unit: six unfolding questions, each with a rationale. Work through each screen as the story unfolds, and check each answer to see the explanation. This case study is free, with no sign-up.
What this case tests
NCLEX heart failure questions: six clinical judgment steps
The case follows one client. The chart gains new entries as the story unfolds, and each question tests one step of the NCSBN Clinical Judgment Measurement Model, in order.
Work through the case on the exam screen
The case opens in the NCLEX-style test interface, one screen at a time, with the chart beside each question. Submit each answer to see the correct answer and the rationale before the story moves on. Your progress is saved in this browser, and the last screen shows your score and where you gained and lost points.
Answer key
Heart Failure case study: answers and rationales
Try the questions first. Open one to read its answer and rationale.
Question 1: Recognize cuesClick to highlight the findings below that require immediate follow-up.
The cues are signs of fluid backing up into the lungs and the venous system: shortness of breath when walking to the bathroom, sleeping upright for 3 nights (orthopnea), crackles in both lung bases, jugular venous distention (JVD), 3+ pitting edema, and a 4 kg (8.8 lb) gain in 1 week, well above the report threshold of 5 lb in 1 week. Each is an early warning cue of decompensation that needs prompt follow-up. The other selectable findings need no immediate action. Full orientation and warm, dry skin argue against the confusion, heavy sweating and cold extremities of acute pulmonary edema, and speaking in full sentences is a normal finding. A hearing aid for long-standing hearing loss is a stable baseline; new ringing in the ears or new hearing loss would matter once IV furosemide starts. Takeaway: in heart failure, dyspnea with usual activity, orthopnea, crackles, JVD, edema and rapid weight gain are congestion cues that need immediate follow-up.
Sources: Open RN Nursing: Health Alterations, 5.8 Heart Failure; US Department of Veterans Affairs: Living with Heart Failure (patient guide); Open RN Nursing Fundamentals (LibreTexts), 15.6 Applying the Nursing Process; StatPearls: Cardiogenic Pulmonary Edema; StatPearls: Creatinine Clearance
Question 2: Analyze cuesFor each finding below, click to specify if the finding is consistent with the disease process of left-sided heart failure or right-sided heart failure. Each finding may support more than 1 disease process.
Left-sided failure backs blood up into the lungs, so its signs are respiratory: dyspnea on exertion, orthopnea (breathless lying flat, so the client sleeps upright) and crackles. Right-sided failure backs blood up into the body's venous system, so its signs are systemic: jugular venous distention (JVD), dependent edema of the legs and ankles, and anorexia, which appears alongside ascites and an enlarged liver. This client shows both patterns, which is common because left-sided failure is a frequent cause of right-sided failure, but each finding listed here belongs to one side only. Takeaway: left-sided failure shows in the lungs; right-sided failure shows in the body.
Sources: Open RN Nursing: Health Alterations, 5.8 Heart Failure; StatPearls: Creatinine Clearance; Open RN Nursing Fundamentals (LibreTexts), 15.6 Applying the Nursing Process
Question 3: Prioritize hypothesesComplete the following sentence by choosing from the lists of options.
The deciding cue is the pulse oximetry reading (SpO₂) of 89% on room air, together with crackles in both lung bases, a respiratory rate of 28 and a heart rate of 112. Fluid backing up into the lungs from left-sided failure is already lowering oxygenation, which puts the client closest to acute pulmonary edema, a breathing emergency. The client is still alert and oriented with warm, dry skin, so the emergency has not yet arrived, but under the ABCs a threat to breathing outranks every other risk on this chart. The other conditions are not supported: the digoxin level is 1.1 ng/mL (reference 0.5–2.0), potassium is 4.0 mEq/L (reference 3.5–5.0) and creatinine is 1.1 mg/dL (reference 0.6–1.2), all within range. Takeaway: when several risks compete, the one that threatens breathing comes first, and a low SpO₂ is the cue that shows it.
Sources: Open RN Nursing: Health Alterations, 5.8 Heart Failure; StatPearls: Cardiogenic Pulmonary Edema; Open RN Nursing Management and Professional Concepts, 2.3 Tools for Prioritizing; DailyMed: digoxin tablets label; Open RN Nursing Pharmacology, 6.7 Cardiac Glycosides; Open RN Nursing Fundamentals (LibreTexts), 15.6 Applying the Nursing Process; StatPearls: Hypokalemia; Open RN Nursing Pharmacology, 6.10 Antihypertensives; StatPearls: Creatinine Clearance
Question 4: Generate solutionsWhich of the following interventions should the nurse include in the client's plan of care? Select all that apply.
The cues are fluid in the lungs (crackles, orthopnea, pulse oximetry reading 89%), fluid in the body (JVD, edema, 4 kg gain) and a plan that will rely on IV diuresis. Sitting upright eases breathing when fluid backs up into the lungs. Oxygen is given as prescribed and titrated to the prescribed pulse oximetry target. A daily weight taken the same way each morning is the main bedside way to track fluid gain or loss. Potassium and creatinine are monitored because loop diuretics waste potassium and excessive diuresis can cause dehydration and kidney injury. Clustering care with rest periods conserves energy. The distractors work against treatment: extra fluid adds to the overload (fluid is limited as ordered, commonly to 2 L/day), a potassium salt substitute adds to the potassium-raising effect of lisinopril, and IV furosemide is pushed slowly over 1–2 minutes because rapid injection risks ototoxicity. Takeaway: care for decompensated heart failure pairs breathing support (upright position, oxygen) with careful fluid removal (daily weights, potassium and kidney checks) and rest.
Sources: StatPearls: Cardiogenic Pulmonary Edema; US Department of Veterans Affairs: Living with Heart Failure (patient guide); Open RN Nursing Fundamentals (LibreTexts), 15.6 Applying the Nursing Process; Open RN Nursing: Health Alterations, 5.8 Heart Failure; DailyMed: furosemide injection label; Open RN Nursing Pharmacology, 6.9 Diuretics; Open RN Nursing Pharmacology, 6.10 Antihypertensives; StatPearls: Creatinine Clearance; StatPearls: Heart Failure (Nursing)
Question 5: Take actionSelect the 3 actions the nurse should take.
Three Day 2 cues drive the actions: potassium has fallen to 3.1 mEq/L (reference 3.5–5.0), digoxin is due at 0900, and the client is weak and lightheaded on standing after a 3,200 mL diuresis. Loop diuretics waste potassium, and low potassium raises the risk of digoxin toxicity, so a potassium below 3.5 mEq/L is reported to the primary health care provider (PHCP) at once, before the next digoxin dose. Lightheadedness after heavy diuresis points to orthostatic hypotension. The nurse measures BP and pulse lying and then standing (a systolic drop of 20 mm Hg or more, a diastolic drop of 10 mm Hg or more, or dizziness on standing is abnormal) and prevents a fall by having the client call for help before getting up. Takeaway: after brisk diuresis, check potassium before giving digoxin and check for orthostatic hypotension before the client walks alone.
Sources: DailyMed: furosemide injection label; Open RN Nursing Pharmacology, 6.9 Diuretics; Open RN Nursing Pharmacology, 6.7 Cardiac Glycosides; DailyMed: digoxin tablets label; Open RN Nursing Fundamentals (LibreTexts), 15.6 Applying the Nursing Process; StatPearls: Hypokalemia; CDC STEADI: Measuring orthostatic blood pressure; StatPearls: Heart Failure (Nursing); DailyMed: potassium chloride concentrate injection label; Open RN Nursing: Health Alterations, 5.8 Heart Failure; StatPearls: Creatinine Clearance
Question 6: Evaluate outcomesFor each assessment finding, click to specify if the finding indicates that the client's condition has improved, not changed, or worsened.
Each Day 3 finding is compared with the Day 1 and Day 2 values. Improved: weight is down from 84 kg on admission and 81 kg on Day 2, showing fluid loss; the pulse oximetry reading is 96% on room air, up from 89% on room air and 94% on oxygen; the client sleeps with 1 pillow instead of upright in a recliner or on 2 pillows, so orthopnea has eased; and crackles have shrunk to a faint area in one base. Not changed: sodium is 137 mEq/L, within the reference range on all 3 days. Worsened: potassium has fallen from 4.0 to 3.1 and now 2.8 mEq/L (reference 3.5–5.0) despite oral replacement, a known effect of loop diuretics, so it is reported to the primary health care provider (PHCP). Takeaway: congestion can clear while diuretic side effects worsen, so evaluation covers both before the plan is judged effective.
Sources: Open RN Nursing: Health Alterations, 5.8 Heart Failure; Open RN Nursing Fundamentals 2e, 4.8 Evaluation; US Department of Veterans Affairs: Living with Heart Failure (patient guide); DailyMed: furosemide injection label; CDC STEADI: Measuring orthostatic blood pressure; Open RN Nursing Fundamentals (LibreTexts), 15.6 Applying the Nursing Process; StatPearls: Hypokalemia; StatPearls: Creatinine Clearance
FAQ
NCLEX heart failure questions: FAQ
What are the signs and symptoms of right-sided heart failure?
What are 5 signs of heart failure?
What are the priority nursing problems in heart failure?
Sources: Open RN Nursing: Health Alterations, 5.8 Heart Failure; NHLBI (NIH): Heart Failure, Symptoms; Open RN Nursing Fundamentals (LibreTexts), 15.6 Applying the Nursing Process; StatPearls: Cardiogenic Pulmonary Edema
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